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JESSE HENRY v. WORKMEN'S COMPENSATION APPEAL BOARD (MAYER POLLOCK CORPORATION) (08/03/88)

decided: August 3, 1988.

JESSE HENRY, DECEASED, ANGELA GERRIS HENRY, WIDOW, PETITIONER
v.
WORKMEN'S COMPENSATION APPEAL BOARD (MAYER POLLOCK CORPORATION), RESPONDENTS



Appeal from the Order of the Workmen's Compensation Appeal Board, in the case of Jesse Henry, Deceased, Angela Gerris Henry, Widow v. Mayer Pollock Corporation, No. A-92695.

COUNSEL

David Freeman, for petitioner.

Steven J. Proctor, Binder, Kalis, Proctor, Breidenbach & Bennett, for respondent.

Judges Craig and Palladino, and Senior Judge Barbieri, sitting as a panel of three. Opinion by Senior Judge Barbieri.

Author: Barbieri

[ 118 Pa. Commw. Page 361]

Angela Gerris Henry (Claimant), widow of Jesse Henry (Decedent), petitions for review of the order of the Workmen's Compensation Appeal Board (Board) dismissing her fatal claim petition. We affirm.

Decedent was employed by Mayer Pollock Corporation (Employer), a wrecking contractor, as a burner. On May 20, 1981, Decedent was burning paint on a job at the Franklin Mint when he inhaled lead fumes from the paint and contracted lead poisoning. Pursuant to a notice of compensation payable, Decedent received benefits for total disability in the amount of $262.00 per week. He successfully underwent chelation treatments until his blood lead level returned to normal. However, Decedent continued to suffer headaches and dizziness which his treating physician attributed to hypertension.

Decedent died on May 2, 1983. Doctor Robert L. Catherman, the deputy medical examiner who performed the autopsy, opined that the cause of death was

[ 118 Pa. Commw. Page 362]

    arteriosclerotic coronary disease and alcoholic cardiomyopathy. Alcoholic cardiomyopathy is a general term used to describe an abnormal enlargement of the heart muscles often found in chronic alcoholics. At the time of his death, Decedent had a blood alcohol level of .18. (Deposition of Doctor Robert L. Catherman taken 7/24/85 at 35.) His gastric or stomach content was .29% alcohol and .28% Elavil, a drug commonly prescribed for depression. (Deposition of Doctor Robert L. Catherman taken 7/24/85 at 35, 37.)

It is uncontradicted that Decedent did not die of lead poisoning. Rather, it is Claimant's theory of the case that Decedent's lead poisoning caused depression, which led to heavy drinking, and that the heavy drinking caused or aggravated Decedent's alcoholic cardiomyopathy, which in turn was a substantial cause of his death. The referee rejected this chain of causation as too tenuous and the Board affirmed.

Claimant's case is supported by her testimony that Decedent had been a social drinker before his incident of lead poisoning but began to drink as much as a fifth of liquor a day six or seven months before he died. However, the referee chose to accept the opinion of Doctor Catherman that alcoholic cardiomyopathy requires many years of excessive abuse of alcohol, and that the degree of enlargement and damage to Decedent's heart was not consistent with alcohol abuse of only six or seven months duration, or even of two years duration. (Deposition of Doctor Robert L. Catherman taken 7/24/85 at 15, 16.) Consequently, the referee found that Decedent's alcohol abuse predated his lead poisoning attack by several years.

Claimant argues that the referee erred in applying the substantial contributing factor test of McCloskey v. Workmen's Compensation Appeal Board (J.H. France ...


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